Laminated root rot also known as yellow ring rot is caused by the fungal pathogen "Phellinus weirii". Laminated root rot is one of the most damaging root disease amongst conifers in northwestern America and true firs, Douglas-fir, Mountain hemlock, and Western hemlock are highly susceptible to infection with "P. weirii". A few species of plants such as Western white pine and Lodgepole pine are tolerant to the pathogen while Ponderosa pine is resistant to it. Only hardwoods are known to be immune to the pathogen.

The disease can infect trees as young as 6 years-old, and infects trees throughout their lifespan. Diagnostic symptoms include crown yellowing and thinning, a distress crop of cones, red brown stained outer heartwood, and laminate decay (decay that separates along annual rings). The disease tends to occur in patches due to a primarily short range spread mechansism. Infected or decayed roots break close to the root collar forming “root balls.” Laminated root rot is frequently detected during ground survey when canopy openings and standing dead and fallen trees are observed. Signs of laminated root rot include the setal hyphae (tiny hairlike hyphae) between sheets of decomposing wood and also buff-colored mycelium on the outside of the roots.

Armillaria root rot is a fungal root rot caused by several different members of the genus "Armillaria". The symptoms are variable depending on the host infected, ranging from stunted leaves to chlorotic needles and dieback of twigs and branches. However, all infected hosts display symptoms characteristic of being infected by a white rotting fungus. The most effective ways of management focus on limiting the spread of the fungus, planting resistant species, and removing infected material. This disease poses a threat to the lumber industry as well as affecting recreational areas.

Common spot of strawberry is one of the most common and widespread diseases of strawberry. Common spot of strawberry is caused by the fungus Mycosphaerella fragariae (imperfect stage is "Ramularia tulasnei"). Symptoms of this disease first appear as circular, dark purple spots on the leaf surface. "Mycosphaerella fragariae" is very host specific and only infects strawberry.

Mycosphaerella fragariae is a species from family Mycosphaerellaceae.

Ascochyta blights occur throughout the world and can be of significant economic importance. Three fungi contribute to the ascochyta blight disease complex of pea ("Pisum sativum"). "Ascochyta pinodes" (sexual stage: "Mycosphaerella pinodes") causes Mycosphaerella blight. "Ascochyta pinodella" (synonym: "Phoma medicaginis" var. "pinodella") causes Ascochyta foot rot, and "Ascochyta pisi" causes Ascochyta blight and pod spot. Of the three fungi, "Ascochyta pinodes" is of the most importance. These diseases are conducive under wet and humid conditions and can cause a yield loss of up to fifty percent if left uncontrolled. The best method to control ascochyta blights of pea is to reduce the amount of primary inoculum through sanitation, crop-rotation, and altering the sowing date. Other methods—chemical control, biological control, and development of resistant varieties—may also be used to effectively control ascochyta diseases.

Certain techniques can be used to determine which pathogen is causing disease. One standard technique for distinguishing strains is microscopy. Under a microscope, "M. pinodes" can be diagnosed by the presence of pseudothecia. "P pinodella" can be diagnosed by the size of conidia produced. "P. pinodella" produces conidia that are smaller than the conidia of "M. pinodes" or "A. pisi". "A. pisi" can be diagnosed by the color of the conidia. In comparison to the light colored, buff spore masses of "M. pinodes" and "P. pinodella" produced on oatmeal agar, "A. pisi" spores masses are carrot red.

Other techniques for diagnosis involve serological assays, isoenzyme analysis, restriction fragment length polymorphisms (RFLPs), random amplified polymorphic DNA (RAPD) assays, and by using monoclonal antibodies.

Bacterial soft rots are caused by several types of bacteria, but most commonly by species of gram-negative bacteria, "Erwinia", "Pectobacterium", and "Pseudomonas". It is a destructive disease of fruits, vegetables, and ornamentals found worldwide, and effects genera from nearly all the plant families. The bacteria mainly attack the fleshy storage organs of their hosts (tubers, corms, bulbs, and rhizomes), but they also affect succulent buds, stems, and petiole tissues. With the aid of special enzymes, the plant is turned into a liquidy mush in order for the bacteria to consume the plant cell's nutrients. Disease spread can be caused by simple physical interaction between infected and healthy tissues during storage or transit. The disease can also be spread by insects. Control of the disease is not always very effective, but sanitary practices in production, storing, and processing are something that can be done in order to slow the spread of the disease and protect yields.

Dead arm, sometimes grape canker, is a disease of grapes caused by a deep-seated wood rot of the arms or trunk of the grapevine. As the disease progresses over several years, one or more arms may die, hence the name "dead arm". Eventually the whole vine will die. In the 1970s, dead-arm was identified as really being two diseases, caused by two different fungi, "Eutypa lata" and "Phomopsis viticola" (syn. "Cryptosporella viticola").

Beet vascular necrosis and rot is a soft rot disease caused by the bacterium Pectobacterium carotovorum" subsp. "betavasculorum, which has also been known as "Pectobacterium betavasculorum" and "Erwinia carotovora" subsp. "betavasculorum". It was classified in the genus "Erwinia" until genetic evidence suggested that it belongs to its own group; however, the name Erwinia is still in use. As such, the disease is sometimes called Erwinia rot today. It is a very destructive disease that has been reported across the United States as well as in Egypt. Symptoms include wilting and black streaks on the leaves and petioles. It is usually not fatal to the plant, but in severe cases the beets will become hollowed and unmarketable. The bacteria is a generalist species which rots beets and other plants by secreting digestive enzymes that break down the cell wall and parenchyma tissues. The bacteria thrive in warm and wet conditions, but cannot survive long in fallow soil. However, it is able to persist for long periods of time in the rhizosphere of weeds and non-host crops. While it is difficult to eradicate, there are cultural practices that can be used to control the spread of the disease, such as avoiding injury to the plants and reducing or eliminating application of nitrogen fertilizer.

Dead arm is a disease that causes symptoms in the common grapevine species, "vitis vinifera", in many regions of the world. This disease is mainly caused by the fungal pathogen, "Phomopsis viticola", and is known to affect many cultivars of table grapes, such as Thompson Seedless, Red Globe, and Flame Seedless. Early in the growing season, the disease can delay the growth of the plant and cause leaves to turn yellow and curl. Small, brown spots on the shoots and leaf veins are very common first symptoms of this disease. Soil moisture and temperature can impact the severity of symptoms, leading to a systemic infection in warm, wet conditions. As the name of this disease suggests, it also causes one or more arms of the grapevine to die, often leading to death of the entire vine.

Sudden Death Syndrome (SDS) in Soybean plants quickly spread across the southern United States in the 1970s, eventually reaching most agricultural areas of the US. SDS is caused by a Fusarium fungi, more specifically the soil borne root pathogen "Fusarium virguliforme," formerly known as "Fusarium solani" f. sp. "glycines"."." Losses could exceed hundreds of millions of dollars in US soybean markets alone making it one of the most important diseases found in Soybeans across the US

The symptoms of Cherry X disease vary greatly depending on the host. On cherry hosts symptoms can usually first be seen on the fruits, causing them to be smaller in size with a leathery skin. Pale fruit is common at harvest time. It is common for symptoms to first be seen in a single branch. The branch may lose its older leaves, and the leaves tend to be smaller with a bronzed complexion.

The rootstock that the cherry is grafted onto can play a significant role in the disease symptoms seen. Rootstocks of Mahaleb cherry exhibit different symptoms from stocks of Colt, Mazzard, or Stockton Morello. When the scion is grafted onto Mahaleb, symptoms consistent with Phytophthora root rot can be seen. To distinguish between root rot and x-disease the wood under the bark at the graft union should be examined. If it is x-disease the wood at the union will have grooves and pits this causes a browning of the phloem and shows the cells in decline. This rapid decline is caused by the rootstock cells near the graft union dying in large quantities. Foliage begins to turn yellow and the curl upward and inward toward the leaf midrib. Trees infected with Mahaleb rootstock die by late summer or early the following year.

When Cherries are grafted onto Colt, Mazzard, or Stockton Morello rootstocks, there is a different range of symptoms. Affected leaves are smaller than normal and the foliage may be sparse. Dieback of shoot tips is common as the disease progresses. Fruit on branches are smaller, lighter, pointed, low sugar content, poor flavor, and a bitter taste.

Peaches are the next most common economic fruit host of the X-disease. Symptoms can be seen after about two months single branches will begin to show symptoms of their individual leaves. These leaves curl up and inward with irregular yellow to reddish-purple spots. These spots can drop out leaving “shotholes”. Leaves that are affected by the disease will fall prematurely. After 2–3 years the entire tree will show symptoms.

This disease affects strawberry plant foliage causing purple spots ⅛ to ¼ inches across on the upper side of the leaves. At first, the whole spot is purple but as the disease matures the center of the leaf spots become tan or gray, then almost white. When numerous spots merge foliage death can occur; this can stunt or kill infected plants when severe.

On petioles, stolons, calyxes, and fruit trusses, elongated lesions may form and interfere with water transport in the plant, weakening the plant and making it more susceptible to invasion by a secondary organism.

Most of the SDS symptoms can be confused with other factors like nutrient deficiencies and some other diseases like brown stem rot and stem canker. Usually the first symptom seen is interveinal chlorosis, which is the yellowing of the plant material between the leaf veins. When leaves begin to die, puckering and mottling can also be observed along with the chlorosis. As severity increases, necrosis (death of cells) occurs and eventually these leaves will fall off, leaving only petioles left on the stem. If the conditions are right (cool and wet), these symptoms can appear suddenly, causing large yield reductions. Normally, this is seen in mid or late July around the time of flowering and pod production.

In addition to foliar symptoms, the stem of the soybean plant can show symptoms as well. If a soybean stem with SDS is split, the pith will be visibly white while the cortical tissue around the pith will be tan to light brown in color. If the pith is brown in color (or if the whole stem looks brown on the inside), it is likely that the plant has brown stem rot, rather than SDS

Along with the above ground foliar and stem symptoms, the roots usually show some kind of rotting and decrease in vigor compared to other healthy soybean roots. If soil conditions are moist, roots are also likely to show blue masses of spores (macroconidia) around the taproot just below the soil surface. Blue fungal masses, found along with the foliar and stem symptoms, are strong diagnostic indicators for SDS

Cherry X disease also known as Cherry Buckskin disease is caused by a plant pathogenic phytoplasma. Phytoplasma's are obligate parasites of plants and insects. They are specialized bacteria, characterized by their lack of a cell wall, often transmitted through insects, and are responsible for large losses in crops, fruit trees, and ornamentals. The phytoplasma causing Cherry X disease has a fairly limited host range mostly of stone fruit trees. Hosts of the pathogen include sweet/sour cherries, choke cherry, peaches, nectarines, almonds, clover, and dandelion. Most commonly the pathogen is introduced into economical fruit orchards from wild choke cherry and herbaceous weed hosts. The pathogen is vectored by mountain and cherry leafhoppers. The mountain leafhopper vectors the pathogen from wild hosts to cherry orchards but does not feed on the other hosts. The cherry leafhopper which feeds on the infected cherry trees then becomes the next vector that transmits from cherry orchards to peach, nectarine, and other economic crops. Control of Cherry X disease is limited to controlling the spread, vectors, and weed hosts of the pathogen. Once the pathogen has infected a tree it is fatal and removal is necessary to stop it from becoming a reservoir for vectors.

There are a variety of hosts including but not limited to; banana, beans, cabbage, carrot, cassava, coffee, corn, cotton, onion, other crucifers, pepper, potato, sweet potato and tomato. For each host there are different symptoms displayed. Most symptoms are along the lines of watery and soft decay of the tissue. Cabbage and crucifers' symptoms start where the tissue makes contact with the soil. Often there is a change in color and in the case of a carrot, the whole taproot can be decayed leaving just the epidermis. Sweet potatoes show clear lesions that grow rapidly leaving a recognizable watery and soft, oozy tissue where only the peel remains intact.

Potatoes experience a cream to tan colored tuber that becomes very soft and watery. A characteristic black border separates the diseased area and the healthy tissue. Only when the secondary organism invades the infected tissue does that decay become slimy with a foul odor. Like the carrot, the whole tuber can be consumed leaving just the epidermis in the soil. The foliage becomes weak and chlorotic with upward turned leaves and lesions on the stem. The stem also rots and becomes mushy with its colorless or brown lesions.

Symptoms can be found on both beet roots and foliage, although foliar symptoms are not always present. If present, foliar symptoms include dark streaking along petioles and viscous froth deposits on the crown which are a by-product of bacterial metabolism. Petioles can also become necrotic and demonstrate vascular necrosis. When roots become severely affected, wilting also occurs. Below ground symptoms include both soft and dry root rot. Affected vascular bundles in roots become necrotic and brown, and tissue adjacent to necrosis becomes pink upon air contact. The plants that do not die completely may have rotted-out, cavernous roots.

Various pathogens can cause root rot in beets; however the black streaking on petioles and necrotic vascular bundles in roots and adjacent pink tissue help to distinguish this disease from others such as Fusarium Yellows. Additionally, sampling from the rhizosphere of infected plants and serological tests can confirm the presence of "Erwinia caratovora" subs.

Root rot is a condition found in both indoor and outdoor plants, although more common in indoor plants with poor drainage. As the name states, the roots of the plant rot. Usually, this is a result of overwatering. In houseplants, it is a very common problem, and is slightly less common in outdoor plants. In both indoor and outdoor plants, it is usually lethal and there is no effective treatment.

The excess water makes it very difficult for the roots to get the air that they need, causing them to decay. To avoid root rot, it is best to only water plants when the soil becomes dry, and to put the plant in a well-drained pot. Using a heavy soil, such as one dug up from outdoors can also cause root rot.

Many cases of root rot are caused by members of the water mould genus "Phytophthora"; perhaps the most aggressive is "P. cinnamomi". Spores from root rot causing agents do contaminate other plants, but the rot cannot take hold unless there is adequate moisture. Spores are not only , but are also carried by insects and other arthropods in the soil.

A plant with root rot will not normally survive, but can often be propagated so it will not be lost completely. Plants with root rot should be removed and destroyed.

Verticillium wilt is a wilt disease of over 350 species of eudicot plants caused by six species of Verticillium genus, "V. dahliae", "V. albo-atrum", "V. longisporum", V. nubilum, V. theobromae and

V. tricorpus. (See, for example, Barbara, D.J. & Clewes, E. (2003). "Plant pathogenic Verticillium species: how many of them are there?" Molecular Plant Pathology 4(4).297-305. Blackwell Publishing.) Many economically important plants are susceptible including cotton, tomatoes, potatoes, oilseed rape, eggplants, peppers and ornamentals, as well as others in natural vegetation communities. Many eudicot species and cultivars are resistant to the disease and all monocots, gymnosperms and ferns are immune.

Symptoms are superficially similar to "Fusarium" wilts. There is no chemical control for the disease but crop rotation, the use of resistant varieties and deep plowing may be useful in reducing the spread and impact of the disease.

Physiological plant disorders are caused by non-pathological conditions such as poor light, adverse weather, water-logging, phytotoxic compounds or a lack of nutrients, and affect the functioning of the plant system. Physiological disorders are distinguished from plant diseases caused by pathogens, such as a virus or fungus. While the symptoms of physiological disorders may appear disease-like, they can usually be prevented by altering environmental conditions. However, once a plant shows symptoms of a physiological disorder it is likely that that season’s growth or yield will be reduced.

Because this disease is caused by multiple species within the genus "Armillaria", it has an extremely broad host range. Hundreds of trees and shrubs are susceptible to root rot to varying degrees. In fact, the only two genera of tree known to be resistant to Armillaria root rot are larch and birch. Further investigation is being conducted for additional species, but at this time there is no further evidence leading to the belief that more exist.

As a result of the multitude of possible hosts, symptoms also range a great deal from one infection to another. All infected hosts do however display symptoms common to white rotting fungi. These include light or bleached wood as a result of the degradation of essential cell wall compounds such as lignin and hemicellulose. In addition to these, the host will show above-ground symptoms due to fungal infection of the vasculature. These include chlorosis in the needles as well as dieback of twigs and branches. The extent of these symptoms varies with the degree of infection as well as the susceptibility of the host. In addition to these symptoms, the trunks of conifers will also exude excess resin in a process known as resinopsis which results in a layer of resin, debris, and fungal tissue forming around infected roots. Deciduous trees occasionally develop sunken cankers but most often fail to exhibit these symptoms on the trunk, and will instead simply display the other symptoms.

In addition to these symptoms, signs of the infectious organism are very evident in the host. Upon removal of the bark, white mycelial mats are visible along with rhizomorphs, a distinctive reproductive structure. Clusters of mushrooms will also form at the base of the infected tree, indicating an infection. Also, zone lines of melanized fungal cells may be visible within infected wood.

Diseases can have a variety of causes, including bacterial infections from an external source such as "Pseudomonas fluorescens" (causing fin rot and fish dropsy), fungal infections (Saprolegnia), mould infections (Oomycete and "Saprolegnia"), parasitic disorders ("Gyrodactylus salaris", "Ichthyophthirius multifiliis", Cryptocaryon, Oodinium causing velvet disease, "Brooklynella hostilis", head and lateral line erosion, Glugea, "Ceratomyxa shasta", "Kudoa thyrsites", "Tetracapsuloides bryosalmonae", "Ceratomyxa shasta" leeches, nematode, Trematoda, Platyhelminthes and fish louse), viral disorders, metabolic disorders, inappropriate water conditions (insufficient aeration, pH, water hardness, temperature and ammonia poisoning) and malnutrition.

External bacterial infections may cause spots or streaks on the body which appear red or orange Dropsy (bloating) is also a sign of a bacterial infection. "False fungal infections" look like fungus but is actually a bacterial infection known as Columnaris. These symptoms may include a white or gray film on the body.

Noble rot (; ; ; ) is the beneficial form of a grey fungus, "Botrytis cinerea", affecting wine grapes. Infestation by "Botrytis" requires moist conditions. If the weather stays wet, the damaging form, "grey rot," can destroy crops of grapes. Grapes typically become infected with "Botrytis" when they are ripe. If they are then exposed to drier conditions and become partially raisined this form of infection is known as noble rot. Grapes when picked at a certain point during infestation can produce particularly fine and concentrated sweet wine. Wines produced by this method are known as botrytized wines.

Fin rot is a symptom of disease or the actual disease in fish. This is a disease which is most often observed in aquaria and aquaculture, but can also occur in natural populations.

Fin rot can be the result of a bacterial infection ("Pseudomonas fluorescens", which causes a ragged rotting of the fin), or as a fungal infection (which rots the fin more evenly and is more likely to produce a white 'edge'). Sometimes, both types of infection are seen together. Infection is commonly brought on by bad water conditions, injury, poor diet, stress, or as a secondary infection in a fish which is already stressed by other disease.

Fin rot starts at the edge of the fins, and destroys more and more tissue until it reaches the fin base. If it does reach the fin base, the fish will never be able to regenerate the lost tissue. At this point, the disease may begin to attack the fish's body; this is called Advanced Fin and Body rot.

Fin rot is common in bettas due to poor water conditions in pet stores.

"Verticillium" spp. attack a very large host range including more than 350 species of vegetables, fruit trees, flowers, field crops, and shade or forest trees. Most vegetable species have some susceptibility, so it has a very wide host range. A list of known hosts is at the bottom of this page.

The symptoms are similar to most wilts with a few specifics to "Verticillium". Wilt itself is the most common symptom, with wilting of the stem and leaves occurring due to the blockage of the xylem vascular tissues and therefore reduced water and nutrient flow. In small plants and seedlings, "Verticillium" can quickly kill the plant while in larger, more developed plants the severity can vary. Some times only one side of the plant will appear infected because once in the vascular tissues, the disease migrates mostly upward and not as much radially in the stem. Other symptoms include stunting, chlorosis or yellowing of the leaves, necrosis or tissue death, and defoliation. Internal vascular tissue discoloration might be visible when the stem is cut.

In "Verticillium", the symptoms and effects will often only be on the lower or outer parts of plants or will be localized to only a few branches of a tree. In older plants, the infection can cause death, but often, especially with trees, the plant will be able to recover, or at least continue living with the infection. The severity of the infection plays a large role in how severe the symptoms are and how quickly they develop.